![]() ![]() However, the total number of nerve endings was lower in the subacromial impingement group than in the fracture group at all ages. When there is a sudden joint movement it sends a strong signal. Adapts slowly, then transmits a steady signal. RUFFINI ENDINGS SKINThe data from the fracture and subacromial impingement groups were not analyzed statistically because of the difference in the number of specimens in each group. What do the Ruffini endings do Most studied answer They are in deep layers of skin and joint capsules. RUFFINI ENDINGS FREEThe number of Pacinian corpuscles, Ruffini receptors, Golgi tendon organ-like receptors, and free nerve endings of typical morphology tended to decrease with age. The Merkel cells are located at the border between the dermis and epidermis and are specialized to detect edges and points. The Meissner corpuscles are stimulated by skin motion and are located in the epidermis layer. The total number of nerve endings decreased with age in both groups. Ruffini endings detect skin stretch and are also located within the dermis layer of the skin. Age-related changes in the number of nerve endings were compared in each group. The resected coracoacromial ligaments (23 specimens) were divided into two groups based on the disorder that required surgery: a fracture group without subacromial impingement (5 specimens) and a group with subacromial impingement (18 specimens). Free nerve endings were found sporadically on the surface of the subacromial bursal side in the ligament. These typical, non-typical, and unclassifiable nerve endings were all widely distributed on the surface of the subacromial side at the insertion into the acromion and around the coracoid process in the ligament. This type of nerve ending responds to stretch and is important in changes in finger position and movement allowing alterations in grip. There were also unclassifiable nerve endings with a morphology which did not fit any of the above categories. Ruffini corpuscles- spindle shaped nerve endings located in dermis and subcutaneous tissue of the skin. In addition, non-typical Pacinian corpuscles and Ruffini receptors with non-typical morphology were present. Meissner corpuscles and Merkel discs are located within the superficial skin. The coracoacromial ligament was found to contain four types of typical nerve endings: Pacinian corpuscles, Ruffini receptors, Golgi tendon organ-like receptors, and free nerve endings. These findings indicate that the development of periodontal Ruffini endings is regulated by trkB-dependent and CR-coexpressing neurons.We performed gold chloride staining of coracoacromial ligaments removed at operation, observed the peripheral nerve endings in the ligament, and studied their distribution and changes with aging in order to clarify the proprioception of the shoulder joint. The Ruffini ending or Ruffini corpuscle is a class of slowly adapting mechanoreceptor thought to exist only in the glabrous dermis and subcutaneous tissue of humans. Compared with trkA and trkC knockouts, the proportion of CR-positive neurons in mandibular and maxillary regions of the trigeminal ganglion of trkB knockout mice is decreased. The lamellar corpuscles (also known as Pacinian corpuscles) in the skin and fascia detect rapid vibrations (of about 200300 Hz). The Merkel nerve endings (also known as Merkel discs) detect sustained pressure. Only free nerve endings were observed in tissue of trkB knockout mice. The bulbous corpuscles (also known as Ruffini endings) detect tension deep in the skin and fascia. However, Ruffini endings were not present in the periodontal ligament of trkB knockout mice. PGP 9.5-positive fibers in the incisal periodontal ligaments of trkA and trkC knockout mice form Ruffini endings distinguished by dendritic ramifications and branches. Innervation patterns of PGP 9.5- and CGRP-immunoreactive fibers were examined in the periodontal ligament of the knockout mice. The periodontal Ruffini endings in the wild-type mice incisor ligament were classified into two types: type I with ruffled outlines, and type II with a smooth outline. To clarify the role of neurotrophin receptors in the development of Ruffini endings, periodontal ligaments and trigeminal ganglia of trkA, trkB, and trkC knockout mice were immunostained for protein gene product 9.5 (PGP 9.5), calcitonin gene-related peptide (CGRP), parvalbumin (PV), and calretinin (CR). Illustrations pour ruffini endings Vecteurs libres de droits Vous ĂȘtes ici : Accueil. ![]()
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